By Martha Taylor Sarno
With chapters containing as much as 50 percentage new insurance, this ebook offers a radical replace of the newest study and improvement within the zone of bought aphasia. insurance comprises the indicators of aphasia, evaluate, neuropsychology, the explicit linguistic deficits linked to aphasia, similar issues, restoration, and rehabilitation. This accomplished compilation, written via probably the most a professional staff within the box, offers an authoritative textual content and reference for graduate scholars, clinicians, and researchers.
- Chapters comprise as much as 50 percentage new coverage
- Provides replace of contemporary examine within the field
- Includes writings through the main an expert employees within the field
- Comprehensive, exhaustive reference tool
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Additional info for Acquired Aphasia, Third Edition
Davis, K. R. (1978). Broca's aphasia: Pathologic and clinical. Neurology, 28, 311-324. Mohr, J. , Watters, W. , & Duncan, G. W. (1975). Thalamic hemorrhage and aphasia. Brain and Language, 2, 3-17. Naeser, M. , Alexander, M. , Levine, H. , Laughlin, S. , & Geschwind, N. (1982). Aphasia with predominantly subcortical lesion sites: Description of three capsular/putaminal aphasia syndromes. Archives of Neurology (Chicago), 39, 2-14. Naeser, M. , & Hayward, R. W. (1978). Lesion localization in aphasia with cranial compound tomography and the Boston Diagnostic Aphasia Exam.
Broca's aphasia: Pathologic and clinical. Neurology, 28, 311-324. Mohr, J. , Watters, W. , & Duncan, G. W. (1975). Thalamic hemorrhage and aphasia. Brain and Language, 2, 3-17. Naeser, M. , Alexander, M. , Levine, H. , Laughlin, S. , & Geschwind, N. (1982). Aphasia with predominantly subcortical lesion sites: Description of three capsular/putaminal aphasia syndromes. Archives of Neurology (Chicago), 39, 2-14. Naeser, M. , & Hayward, R. W. (1978). Lesion localization in aphasia with cranial compound tomography and the Boston Diagnostic Aphasia Exam.
A few have acute psychotic states and no macroscopically detectable brain disease, although they may suffer from profound changes in neurotransmitter systems innervating certain regions of the brain. Also not aphasic are patients with aphonia that may result from diseases of the larynx and pharynx. They are mute, in the narrow sense of the word, and are suffering from an impediment in their phonatory apparatus that prevents them from speaking. They should be able to comprehend language (and indicate so by nodding or pointing responses), and they should be able to turn their thoughts into language by writing, in addition to being able to mouth words.