By Heatwole, H. & J. W. Wilkinson (eds.)
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Additional resources for Amphibian biology. Volume 8. Amphibian decline: Diseases, parasites, maladies and pollution
Family Iridoviridae. Pp. 167-182 i n "Virus Taxonomy, Seventh Report of the International Committee on Taxonomy of Viruses", ed by M. H. V. Van Regenmortel, C. M. Fauquet, D. L. Bishop, E. B. Carsten, M. K. Estes, S. M. Lemon, J. Maniloff, M. A. Mayo, D. J. McGeoch, C. R. Pringle, and R. B. Wickner. Academic Press: San Diego, USA. Willis D. , Goorha, R. and Chinchar, V. , 1985. Macromolecular synthesis in cells infected by frog virus 3. Curx Topics in Microbiology and Immunology 116: 77-106. , Bullock, G.
Wildl. Disease 43: 525-532. Greer, A. L. and Collins, J. , 2008. Habitat fragmentation as a result of biotic and abiotic factors controls pathogen transmission throughout a host population. J. Anim. Ecol. 77: 364-369. Greer, A. , Berrill, M. and Wilson, F! , 2005. Five amphibian mortality events associated with ranavirus infection in south central Ontario, Canada. Diseases of Aquatic Organism 67: 9-14. Greer, A. , Briggs, C. J. and Collins, J. , 2008. Testing a key assumption of host-pathogen theory: density and disease transmission.
It caused epidemic waves of high mortality as it spread through na'ive populations. Once introduced, it persists and acts as an endemic pathogen. If populations survive, their mortality rate appears to be reduced suggesting there is selection for resistance (Retallick et al. 2004; McDonald et al. 2005). Subsequent mortality appears to be the result of interaction between the frogs and environment, particularly temperature (Berger 2001; Woodhams et al. 2003; Berger et al. 2004; McDonald et al. 2005).